GliSODin® abrogates asthma features and reduces CD4+ T-cell polarization and reactivation. Taken together, these data suggest that GliSODin® could be used for the management of asthma symptoms.
Clinical research and scientific evidence is presented to demonstrate that gliadin protects SOD during passage through the stomach, thus allowing absorption of the SOD enzyme once inside the intestine.
According to the results of this study, it was
concluded that supplementation with GliSODin can protect athletes from muscle damage and
decrease inflammation caused by intensive physical activity and have some positive influence on
the sports performance of elite rowers
The results showed that enteral administration of CME-gliadin of 1 IU/day and 5 IU/day
significantly increased SOD levels based on examination after 14 and 28 days. Also, it significantly decreased
MDA (p < 0.001), TNF-α (p < 0.001), and lactate levels in rats induced by sepsis
Melon GliSODin® tended to improve the subjective symptoms of participants who had knee or lower back pain or discomfort. Melon GliSODin® administration may help to prevent the progression of locomotive syndrome. Future studies involving larger sample sizes and more stringent randomization protocols are needed to determine differences between the placebo and Melon GliSODin® groups
After 24 weeks, a greater improvement in VES was observed in the GPSOD group (19.85%; SE 4.63, P < 0.0001) compared with the placebo group (8.83%; SE 4.72, P = 0.0676). Tolerance was good in both groups. No related side-effect was reported. Conclusions : The use of GP-SOD appears to be a useful add-on to phototherapy in the treatment of vitiligo patients
Administration of superoxide dismutase (SOD) increased apoptosis in a prostate cancer cell line (PC-3) through the increased expression of caspase-3. Superoxide dismutase (SOD) can be onsidered as a therapy for late-stage prostate cancer that had been progressed to hormone resistant and metastasized and promote apoptosis in those prostate cancer cells
The results of this study provide in vivo evidence that GliSODin reduced the AOM-induced colon cancer in rats, through their potent antioxidant activities.
Cucumis melo–gliadin inhibits endometriotic implants extension by decreasing MDA serum and TNF-α expression and resulted in lower VEGF expression compare with the control
This study aimed to assess the antioxidant activity of Melon GliSODin and its effectiveness in preventing NASH.
