Orally supplemented GliSODin has been shown scientifically to offer a therapeutic means for the prevention and treatment
of many conditions associated with inflammation and oxidative stress
Antioxidants’ are all individual in their properties; in their cellular compartmentalisation, in their ability to cross cell membranes, in the speed with which they undergo chemical reactions (kinetics), in their solubility, in their bioavailability, in their interactions with other molecules and in their ability to act as messengers, sending signals which initiate critical processes.
These finding suggests that GliSODin prevents stress-induced impairment of cognitive function and maintains neurogenesis in the hippocampus through antioxidant activity.
Antioxidant supplementation resulted in an increase in SOD levels, confirming the oral bioavailability of the compound in FIV-infected cats.
The extreme toxicity of oxygen is related to its unique capacity of generating free radicals, which have long been recognized as harmful compounds on biological molecules.
The photoprotective effect of Glisodin® was studied by Minimal Erythematous Dose (MED) before and after treatment and its efficacy as a treatment of actinic erythema was assessed by biometrological measurements in 49 healthy subjects .
Pre-treatment with the oral melon extract/gliadin may be a therapeutic option to reduce oxidative cell injury affiliated with aortic cross-clamping
Supplementation with GliSODin, a vegetal SOD associated with gliadin, was effective in controlling the thickness of the carotid artery intima and media layers as measured by ultrasonography-B.
Patients with acquired immunodeficiency syndrome (AIDS) present a variety of pathologic alterations that are related to oxidative stress.
Clinical trial using healthy volonteers confirmed the preservation of the antioxydant activity of the SOD enzyme after oral administration; an action moreover combined with anti-inflammatory and immunomodulatory properties.
